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1.0 Introduction Methamphetamine referred to as "meth" ; is a powerful, highly toxic, addictive drug that is illegally "cooked" in makeshift labs. Meth can be found in the form of pills, capsules, powder or chunks. It can be smoked, snorted, injected or eaten. Meth is also called crank, speed, crystal or ice. Meth laboratories have been a growing problem throughout Colorado and across the United States. In Colorado alone, the number of meth lab seizures reported by the Colorado Bureau of Investigation has increased dramatically over the past several years: 150 in 1999, 264 in 2000, 452 in 2001, and the number exceeded 700 in 2002 CDPHE 2003 ; . In accordance with House Bill 04-1182, the Colorado Department of Public Health and Environment CDPHE ; is developing regulations for the cleanup of properties used as meth labs. One goal is to develop re-occupation standards that will protect the occupants mainly children ; from residual chemicals left from the production of illicit drugs. Methamphetamine production is associated with the release of numerous chemicals, such as volatile organic compounds VOCs ; , acids, bases, metals and chemical salts, in addition to methamphetamine itself. Specific chemical residues may vary depending on the cooking process that is utilized. Airborne contaminants are absorbed or deposited onto surfaces such as rugs, furniture, drapes, and walls and may also enter and contaminate heating, ventilation, and air conditioning HVAC ; systems. Chemical spills are not uncommon and may also impact residential surfaces. Presence of these chemicals may pose a health-risk to residents who reoccupy these structures after seizure CDPHE 2003 ; . In order to determine acceptable risk-based concentrations for meth lab related chemicals, CDPHE has previously reviewed human exposure reference values for chemicals commonly associated with meth production CDPHE 2003 ; . Many of these chemicals are well studied and have established concentrations that are thought to be protective under a residential exposure scenario. However, to date, no health-based value for methamphetamine has been developed. Several states have established cleanup standards specifically for the residue of methamphetamine. After communicating with some of these state health departments, it was learned that these levels are not health-based, but are rather based on analytical detection limits. Health-based values could not be established due to deficiencies in the toxicity database. These current meth cleanup levels are instead based on what is believed to be conservative and protective, while at the same time achievable by clean-up contractors. Although numerous states have adopted these detection based cleanup standards for methamphetamine, none have tried to correlate these levels to known health-effect-based concentrations. This paper attempts to reconcile what is known about methamphetamine health effects with those levels currently being used as cleanup standards in order to support selection of a Colorado standard for methamphetamine cleanup. Analytical methods are constantly being refined and detection limits lowered. Simply setting a cleanup standard based on the current detection limit does not provide information on potential health effects. The information summarized in this document will provide Colorado with a balanced approach for weighing these lower detection limits against practicability and cost considerations.
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Methamphetamine precursors, methcathinone, or tetrahydrocannabinols or any controlled substance analog of any of these substances together with any compound, mixture, diluent, plant material or other substance mixed or combined with the controlled substance or, controlled substance analog, or methamphetamine precursor. In addition, in determining amounts under subs. 1 ; h ; and 1m ; h ; , the amount of tetrahydrocannabinols means anything included under s. 961.14 4 ; t ; and includes the weight of any marijuana. SECTION 23. 961.437 title ; of the statutes is amended to read: 961.437 title ; Possession and disposal of Materials used in and waste produced from manufacture of methamphetamine. SECTION 24. 961.437 1 ; title ; of the statutes is created to read: 961.437 1 ; title ; DEFINITIONS. SECTION 25. 961.437 2 ; of the statutes is renumbered 961.437 3m ; a ; . SECTION 26. 961.437 2m ; of the statutes is created to read: 961.437 2m ; MATERIALS USED IN MANUFACTURE OF METHAMPHETAMINE. Except as authorized by this chapter, any person who possesses a methamphetamine precursor, red phosphorus, lithium metal, sodium metal, iodine, anhydrous ammonia, pressurized ammonia, or liquid nitrogen with intent to manufacture methamphetamine is guilty of a Class H felony. Possession of more than 24 grams of methamphetamine precursors shall be prima facie evidence of intent to manufacture methamphetamine.
Table 2. Detection of tlh and tdh genes in Vibrio parahaemolyticus isolates. AP: alkaline phosphatase-labeled probe; DIG: digoxigenin-labeled probe; tlh: thermolabile hemolysin gene; tdh: thermostable direct hemolysin gene Isolate AP-tlh DIG-tlh PCR-tlh DIG-tdh PCR-tdh AP-tdh Environmental 1094 + 1163 + Clinical 2030 2062 2107.
MRNA species from the glyceraldehyde-3-phosphate dehydrogenase multigenic family. Nucleic Acids Res 13: 14311442. Freedman NJ, Lefkowitz RJ 1996 ; Desensitization of G-proteincoupled receptors. Recent Prog Horm Res 51: 319 351. Gainetdinov RR, Wetsel WC, Jones SR, Levin ED, Jaber M, Caron MG 1999 ; Role of serotonin in the paradoxical calming effect of psychostimulants on hyperactivity. Science 283: 397 401. Garrett BE, Holtzman SG 1994 ; D1 and D2 dopamine receptor antagonists block caffeine-induced stimulation of locomotor activity in rats. Pharmacol Biochem Behav 47: 89 94. Gerfen CR, Young WS 1988 ; Distribution of striatonigral and striatopallidal peptidergic neurons in both patch and matrix compartments: an in situ hybridization histochemistry and fluorescent retrograde tracing study. Brain Res 460: 161167. Gerfen CR, Engber TM, Mahan LC, Susel Z, Chase TN, Monsma FJ Jr, Sibley DR 1990 ; D1 and D2 dopamine receptor-regulated gene expression of striatonigral and striatopallidal neurons. Science 250: 1429 1432. Giros B, Jaber M, Jones SR, Wightman RM, Caron MG 1996 ; Hyperlocomotion and indifference to cocaine and amphetamine in mice lacking the dopamine transporter. Nature 379: 606 612. Greengard P, Allen PB, Nairn AC 1999 ; Beyond the dopamine receptor: the DARPP-32 protein phosphatase-1 cascade. Neuron 23: 435 447. Herbert MA, Larson GA, Zahniser NR, Gerhardt GA 1999 ; Agerelated reductions in 3H ; WIN35428 binding to the dopamine transporter in nigrostriatal and mesolimbic brain regions of the Fisher 344 rat. J Pharmacol Exp Ther 288: 1334 1339. Hersch SM, Ciliax BJ, Gutekunst CA, Rees HD, Heilman CJ, Yung KK, Bolam JP, Ince E, Yi H, Levey AI 1995 ; Electron microscopic analysis of D1 and D2 dopamine receptor proteins in the dorsal striatum and their synaptic relationships with motor corticostriatal afferents. J Neurosci 15: 52225237. Herve D, Levi-Strauss M, Marey-Semper I, Verney C, Tassin JP, Glowinski J, Girault JA 1993 ; G olf ; and Gs in rat basal ganglia: possible involvement of G olf ; in the coupling of dopamine D1 receptor with adenylyl cyclase. J Neurosci 13: 22372248. Herve D, Rogard M, Levi-Strauss M 1995 ; Molecular analysis of the multiple Golf alpha subunit mRNAs in the rat brain. Brain Res Mol Brain Res 32: 125134. Ibanez CF, Ernfors P, Persson H 1991 ; Developmental and regional expression of choline acetyltransferase mRNA in rat central nervous system. J Neurosci Res 29: 163171. Jaber M, Robinson SW, Missale C, Caron MG 1996 ; Dopamine receptors and brain function. Neuropharmacology 35: 15031519. Jarvis MF, Williams M 1989 ; Direct autoradiographic localization of adenosine A2 receptors in the rat brain using the A2-selective [ 3H]CGS 21680. Eur J Pharmacol 168: 243246. Jones DT, Reed RR 1989 ; Golf: an olfactory neuron specific-G-protein involved in odorant signal transduction. Science 244: 790 795. Kawaguchi Y, Wilson CJ, Augood SJ, Emson PC 1995 ; Striatal interneurones: chemical, physiological and morphological characterization. Trends Neurosci 18: 527535. Kemp JM, Powell TP 1971 ; The structure of the caudate nucleus of the cat: light and electron microscopy. Philos Trans R Soc Lond B Biol Sci 262: 383 401. Kull B, Svenningsson P, Fredholm BB 2000 ; Adenosine A2A receptors are colocalized with and activate Golf in rat striatum. Mol Pharmacol 58: 771777. Ledent C, Vaugeois JM, Schiffmann SN, Pedrazzi T, El Yacoubi M, Vanderhaeghen JJ, Costentin J, Heath JK, Vassart G, Parmentier M 1997 ; Agressiveness, hypoalgesia and high blood pressure in mice lacking the adenosine A2a receptor. Nature 388: 674 678. Ledent C, Valverde O, Cossu G, Petitet F, Aubert JF, Beslot F, Bohme GA, Imperato A, Pedrazzini T, Roques BP, Vassart G, Fratta W, Parmentier M 1999 ; Unresponsiveness to cannabinoids and reduced addictive effects of opiates in CB1 receptor knock-out mice. Science 283: 401 404. Le Moine C, Bloch B 1995 ; D1 and D2 dopamine receptor gene expression in the rat striatum: sensitive cRNA probes demonstrate prominent segregation of D1 and D2 mRNAs in distinct neuronal populations of the dorsal and ventral striatum. J Comp Neurol 355: 418 426. Le Moine C, Normand E, Guitteny AF, Fouque B, Teoule R, Bloch B 1990 ; Dopamine receptor gene expression by enkephalin neurons in rat forebrain. Proc Natl Acad Sci USA 87: 230 234. Le Moine C, Normand E, Bloch B 1991 ; Phenotypical characterization of the rat striatal neurons expressing the D1 dopamine receptor gene. Proc Natl Acad Sci USA 88: 4205 4209. Levis MJ, Bourne HR 1992 ; Activation of the alpha subunit of Gs in intact cells alters its abundance, rate of degradation, and membrane avidity. J Cell Biol 119: 12971307. Marcotte ER, Sullivan RM, Mishra RK 1994 ; Striatal G-proteins: Ef.
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Ability of wild-type MM-CK. The labeling was highly specific and did not show any affinity for the I-band region. The homologous chimera 2 showed selective I-band-binding ability, known from wild-type BB-CK, without any affinity towards the M-band. Chimera 3 and 4, designed to study the functional role of the exchanged C-terminal CK regions for the isoproteinspecific localization in skeletal muscle, showed also the same binding properties as their parental wild-type CK protein see Table 2 ; . Chimera 3, M-CK with C-terminal part from B-CK, still showed the characteristic M-band-binding pattern as did wild-type MM-CK. This highly specific labeling did not show any affinity for the I-band region in muscle. The homologous chimera 4, B-CK with C-terminal part from M-CK, showed the same I-band-binding properties known from wild-type BB-CK. A minor affinity for the M-band could be observed but only after reference labeling with rhodamine-labeled MM-CK. A final set of chimerae was used to study the functional role of the N-terminal region for the isoprotein-specific localization in skeletal muscle. The resulting labeling patterns of chimerae 5 and 6 is shown in Fig. 5. In contrast to all other chimerae, these two displayed the binding property of the parental CK isoform accounting for the Nterminal region in the chimeric protein. Chimera 5, B-CK with the N-terminal part from M-CK but the entire rest of BCK, showed the strong, highly specific M-band-binding property of wild-type MM-CK without any affinity at all towards the I-band region in muscle. The homologous chimera 6, M-CK with N-terminal part from B-CK but the entire rest of M-CK, showed similar I-band-binding properties as was demonstrated for wild-type BB-CK. The somewhat weaker binding strength of chimera 6, an observation also made with wild-type BB-CK, was again variable and depending on the protein batch used for labeling and cyclosporine.
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Abbreviations: AFREGS, Armed Forces Regression Study; apo A-1, apolipoprotein A-I; ARBITER, Arterial Biology for the Investigation of the Treatment Effects of Reducing Cholesterol; BECAIT, Bezafibrate Coronary Atherosclerosis Intervention Trial; BIP, Bezafibrate Infarction Prevention Study; CAD, coronary artery disease; CDP, Coronary Drug Project; CLAS, Cholesterol Lowering Atherosclerosis Study; CLAS Fem, femoral atherosclerosis group of CLAS; CLAS IMT, carotid ultrasound group of CLAS; DAIS, Diabetes Atherosclerosis Intervention Study; ERASE, Effect of rHDL on Atherosclerosis-Safety and Efficacy Trial; FATS, Familial Atherosclerosis Treatment Study; FIELD, Fenofibrate Intervention and Event Lowering in Diabetes Study; HATS, HDL-Atherosclerosis Treatment Study; HDL-C, high-density lipoprotein cholesterol; HHS, Helsinki Heart Study; ILLUSTRATE, Investigation of Lipid Level Management Using Coronary Ultrasound to Assess Reduction of Atherosclerosis by CETP Inhibition and HDL Elevation Trial; IMT, intima-media thickness; IVUS, intravascular ultrasound; LEADER, Lower Extremity Arterial Disease Event Reduction Trial; LOCAT, Lopid Coronary Angiography Trial; MI, myocardial infarction; NR, not reported; RADIANCE, Rating Atherosclerotic Disease Change by Imaging with a New CETP Inhibitor Trial; SCRIP, Stanford Coronary Risk Intervention Project; TIA, transient ischemic attack; VA-HIT, Veterans Affairs High-Density Lipoprotein Cholesterol Intervention Trial; WHO, World Health Organization. a Death indicates all-cause mortality.
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Mutation that could have reversed the effect of plasmid loss on this isolate. The possibility therefore remains that the acquired resistance to drugs and Hg2 + and the changes in lipid composition are determined by the plasmid. This phenomenon has been observed previously where the presence of colicin 4 ; or resistance 10 ; plasmids not only affected the drug sensitivity of the host, but also mediated substantial changes in the composition of the cell envelope. For example, moving the RP1 plasmid into Pseudomonas aeruginosa has resulted in cells with outer membrane PC levels decreased more than fivefold compared with those of the plasmidless wild type 10 ; . The parallel to the PC-plasmid relationship in R. rubrum is striking. Judging from the drastically altered lipid composition in the cured R. rubrum cells Table 3 ; , membrane modifications could be responsible for the acquired resistance to HgCl2 and cycloserine Fig. 5 ; , just as the altered membranes are believed to have affected the drug sensitivity patterns of P. aeruginosa 10 ; . We previously reported the isolation of plasmid-carrying Psg + revertants arising from plasmidless Psg- ; cultures S. A. Kuhl and D. C. Yoch, Abstr. Annu. Meet. Am. Soc. Microbiol. 1981, H126 p. 134 ; , but they were also extremely rare only two were obtained ; , and the method used to isolate them did not produce any more. We could not be certain, therefore, that these so-called revertants were not, in fact, wild-type contaminants, especially since the DNA of the cured strains from which they appeared to arise did not contain any plasmid sequence homology Fig. 2 ; . The available evidence suggests that the plasmid is involved in the sequence of events resulting in photosynthetic incompetence in the plasmidless R. rubrum mutants, but its exact role in this process is unclear and is currently under investigation and cytarabine.
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Fig. 1. Organization of the adult-rabbit SVZ and its extensions. a ; 3D reconstruction of the SVZ green ; over the ventricles yellow ; . Representative coronal sections 15 ; show the SVZ internal organization: glial densities green ; and PSA-NCAM-immunoreactive chains and masses of cells pink ; . Coronal IIII ; and horizontal IV ; sections revealing PSA-NCAM anterior Ant ; and posterior Post ; chains into the mature brain parenchyma. Their position is indicated by red dots in schematic sections 1 and 5. b Middle ; Distribution of PSA-NCAM masses and chains of cells into pink ; and outside red ; the SVZ. Serial 3D reconstruction of anterior Ant ; and posterior Post ; chains located outside the SVZ area green ; , at the level of the rostral extension RE ; and ventral-lateral extension VLE ; . Chains connected with the dorsal part of the RE are immersed into the white matter of the anterior forcep AF ; of the corpus callosum in b, I, a sagittal section immunostained for PSA-NCAM ; . a, III and IV ; Chains from the VLE, regularly arranged between the external capsule E ; and the striatum S ; . A, amygdala; P, cortex; OV, olfactory ventricle. Bars: a, IIII, 45 m; a, IV, 20 m; b, 80 m.
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C. Ken Williams, MS * , New Jersey State Police Department, South Regional Laboratory, 1101 South Whitehorse Pike, Hammonton, NJ; Amy C. Price, BS, Virginia Division of Forensic Science, 6600 Northside HS Road, 2nd Floor, Roanoke, VA, Claire E. Shepard, MS, 3630 Camp Circle, Crime Scene Unit, Decatur, GA; Sheila M. Estacio, BA * , Office of the Chief Medical Examiner - NYC Office, Department of Forensic Biology, 520 First Avenue, New York, NY; Edward G. Bernstine, MS, PhD * , State Police Crime Laboratory, 702 South Westfield Street, Feeding Hills, MA 01030; Charles H. Dold, JD, MBA * , 13145 92nd Avenue, NE, Kirkland, WA; David L. Exline, MSFS, Chemicon, Inc, 7301 Penn Avenue, Pittsburgh, PA; Eric W. Greenberg, BA, MFS * , 935 Pennsylvania Avenue, NW, Washington, DC; Christopher M. Gojcz, BS * , 28 Atwood Avenue, Pawtucket, RI; Carol Henderson, JD * , Shepard Broad Law Center, Nova Southeastern University, 3305 College Avenue, Ft. Lauderdale, FL; Graham R. Jones, PhD * , Medical Examiner's Office 7007-116 Street Northwest, Edmonton, AB, Canada; Kenneth E. Melson, JD * , United State's Attorney's Office, 2100 Jamieson Avenue, Alexandria, VA; Kathleen J. Reichs, PhD * , UNC-Charlotte, Department of Sociology & Anthropology, Charlotte, NC; Marie Samples, MS * , Office of the Chief Medical Examiner, Department of Forensic Biology, 520 First Avenue, New York, NY; Helena Soomer, DDS * , Department of Forensic Medicine, University of Helsinki, P.O. Box 40, Helsinki, Finland The role of the Young Forensic Scientist has changed drastically over the past few years. The many "forensic science" television shows have caused the public to expect answers in minutes and for laboratories to work miracles. The sensationalism creates its share of backlogs, but it is not the only factor in the changing role. Recent terrorist attacks, and the threat of future ones, have also impacted the future of the young scientist. The pressures placed on forensic science by the public and the constantly growing caseloads have created a need for young forensic scientists to quickly make the transition from school to employment and then to expert. The best preparation for the transition is to be informed of the situation. Focusing on the factors involving both the aspiring and the emerging forensic scientist, the full day program consists of presentations by established members of the forensic science community as well as those that are new to field. The day session concludes with a mock trial that is designed to prepare the attendees for courtroom testimony through demonstration. An evening session has been added to provide a more concentrated focus on those attendees looking to enter the field. Registrants are encouraged to bring copies of their resums in order to have them fine-tuned during an interview and resum workshop. The informal atmosphere and the experiences of the new and established members of the field create an environment that fosters questions and discussion. The program should appeal to individuals with a strong desire to enter the field of forensic science as well as those with a few years of experience and looking to get ahead. The combination of lecture, demonstration and handouts will help to accomplish this goal. Realizing that the knowledge and experience level will vary greatly, the program is designed to touch upon many topics, ranging from the procedure in which one must follow to make a presentation at an Academy meeting to fine-tuning your resum for that very first job search. Emerging Forensic Scientists will share their tales of landing that first job in the field and their involvement with the World Trade Center Disaster. Established members of the field will also be in attendance to share their experiences and to help answer any questions that and cytoxan.
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Nevirapine or efavirenz-containing antiretroviral therapy: role of hepatitis C and B infections. Hepatology 2002; 35: 182-89. Bossi P, Colin D, Bricaire F, Caumes E. Hypersensitivity syndrome associated with efavirenz therapy. Clin Infect Dis 2000; 30: 227-28. Bonnet F, Bonarek M, Morlat P, Mercie P, Dupon M, Gemain MC, Malvy D, Bernard N, Pellegrin JL, Beylot J. Risk factors for lactic acidosis in HIV-infected patients treated with nucleoside reversetranscriptase inhibitors: a case-control study. Clin Infect Dis 2003; 36: 132428. Sitas F, Pacella-Norman R, Carrara H, Patel M, Ruff P, Sur R, Jentsch U, Hale M, Rowji P, Saffer D, Connor M, Bull D, Newton R, Beral V. The spectrum of HIV-1 related cancers in South Africa. Int J Cancer 2000; 88: 489-92. Dal Maso L, Serraino D, Franceschi S. Epidemiology of AIDS-related tumours in developed and developing countries. Eur J Cancer 2001; 37: 1188-201. Puoti M, Bruno R, Soriano V, Donato F, Gaeta GB, Quinzan GP, Precone D, Gelatti U, Asensi V, Vaccher E. Hepatocellular carcinoma in HIVinfected patients: epidemiological features, clinical presentation and outcome. AIDS 2004; 18: 2285-93. Kramer JR, Giordano TP, Souchek J, Richardson P, Hwang LY, El Serag HB. The effect of HIV coinfection on the risk of cirrhosis and hepatocellular carcinoma in U.S. veterans with hepatitis C. J Gastroenterol 2005; 100: 56-63. Raj V, Afdhal NH. Cholangitis in the immunosuppressed patient AIDS cholangiopathy ; . UpToDate 2000; 8. 243. Breitbart W, Rosenfeld BD, Passik SD, McDonald MV, Thaler H, Portenoy RK. The undertreatment of pain in ambulatory AIDS patients. Pain 1996; 65: 243-49. Meyer M. Palliative care and AIDS: 1-pain. Int J STD AIDS 1999; 10: 80-86 and dacarbazine.
If you feel that medical information we have about you is incorrect or incomplete, you may ask us to amend the information. You must provide a reason that supports your request. We may deny your request if the information is not kept by or for the Plan, was not created by us, unless the entity who created the information is not available to make the amendment, or is not part of the information which you would be permitted to inspect or copy, or if the information is accurate and complete. 72 and cycloserine.
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