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Dorsey B, see Hussain A Dorssers LCJ, see Jansen MPHM Dorval M, Guay S, Mondor M, Masse B, Falardeau M, Robidoux A, Deschenes L, Maunsell E. Couples Who Get Closer After Breast Cancer: Frequency and Predictors in a Prospective Investigation, 3588 Dorval T, see van Baren N Dosik M. Withholding Information From Study Participants in Clinical Trials: Ethical Considerations correspondence ; , 4468 Dotan ZA, see Stephenson AJ Dotan ZA, Bianco FJ Jr, Rabbani F, Eastham JA, Fearn P, Scher HI, Kelly KW, Chen H-N, Schoder H, Hricak H, Scardino PT, Kattan MW. Pattern of Prostate-Specific Antigen PSA ; Failure Ditates the Probability of a Positive Bone Scan in Patients With an Increasing PSA After Radical Prostatectomy, 1962 Douer D, Tallman MS. Arsenic Trioxide: New Clinical Experience With an Old Medication in Hematologic Malignancies, 2396 Douglas L, see Canil CM Douglas V, see Gordan LN Douillard J-Y, see Leighl NB Dourthe LM, see Oudard S Doval D, see Ibrahim NK Dowd I, see Baka S Dowell JE, Shen Y, Harford WV, Lai WS. Difficult Diagnostic Cases. Melanoma in African Americans, 3622 Dowell JM, see Reardon DA Dowlati A, see Burris HA III see Spector NL Downey E, see Raja V Downie FP, see Mar Fan HG Downing JR, see Pui C-H Dowsett M, see Gutierrez MC see Smith IE see Urruticoechea A Dowsett M, Cuzick J, Wale C, Howell T, Houghton J, Baum M. Retrospective Analysis of Time to Recurrence in the ATAC Trial According to Hormone Receptor Status: An Hypothesis-Generating Study, 7512 Dowsett M, Ebbs SR, Dixon JM, Skene A, Griffith C, Boeddinghaus I, Salter J, Detre S, Hills M, Ashley S, Francis S, Walsh G, Smith IE. Biomarker Changes During Neoadjuvant Anastrozole, Tamoxifen, or the Combination: Influence of Hormonal Status and HER-2 in Breast Cancer--A Study from the IMPACT Trialists, 2477 Doyle GV, see Cristofanilli M Doyle JJ, Neugut I, Jacobson JS, Grann VR, Hershman DL. Chemotherapy and Cardiotoxicity in Older Breast Cancer Patients: Population-Based Study, 8597 Doyle LA, see Edelman MJ Doyle-Lindrud S, see Goodin S Doz F, see Oyharcabal-Bourden V Drach J, see Raderer M Dragani TA, see Spinola M Dragnev KH, Petty WJ, Shah S, Biddle A, Desai NB, Memoll V, Rigas JR, Dmitrovsky E. Bexarotene and Eriotinib for Aerodigestive Tract Cancer, 8757 Dranitsaris G, see Gainford MC see Verma S Dranoff G. CTLA-4 Blockade: Unveiling Immune Regulation editorial ; , 662 Dravet F, see Rouanet P Drayson MT, see Augustson BM Dreno B, see van Baren N Drenth JPH, see van Kouwen MCA Dressler LG, Berry DA, Broadwater G, Cowan D, Cox K, Griffin S, Miller A, Tse J, Novotny D, Persons DL, Barcos M, Henderson IC, Liu ET, Thor A, Budman D, Muss H, Norton L, Hayes DF. Comparison of HER2 Status.
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`251-Lysergic acid diethylamide `251-LSD ; binds with high affinity to serotonergic sites on rat choroid plexus. These sites were localized to choroid plexus epithelial cells by use of a novel high resolution stripping film technique for light microscopic autoradiography. In membrane preparations from rat choroid plexus, the serotonergic site density was 3100 fmol mg of protein, which is lo-fold higher than the density of any other serotonergic site in brain homogenates. The choroid plexus site exhibits a novel pharmacology that does not match the properties of 5hydroxytryptamine-la 5 HT ; , 5-HTlb, or 5-HT2 serotonergic sites. `251-LSD binding to the choroid plexus site is potently inhibited by mianserin, serotonin, and + ; -LSD. Other serotonergic, dopaminergic, and adrenergic agonists and antagonists exhibit moderate to weak affinities for this site. The rat choroid plexus `251-LSD binding site appears to represent a new type of serotonergic site which is located on non-neuronal cells in this tissue. `"51-Lysergic acid diethylamide "51-LSD ; binds to serotonin 5hydroxytryptamine-2 5HT2 ; receptors in the mammalian brain Engel et al., 1984; Kadan et al., 1984 ; . In a recent autoradiographic study, Nakada et al. 1984 ; noted a high level of "51-LSD binding in the lateral ventricles, which was displaced by ketanserin, a potent serotonin 5HT, ligand. Other investigators have described the binding of 3H-LSD and 3H-serotonin to a particular ventricular structure, the choroid plexus Diab et al., 1971; Meibach et al., 1980; Palacios et al., 1983 ; , but these studies did not describe the pharmacological properties of the choroid plexus binding sites. We decided to investigate ventricular ` * ?LSD-binding sites to determine their cellular location and binding properties. Our results show that binding sites for lz51-LSD are located on epithelial cells of the choroid plexus. These binding sites exhibit a unique serotonergic pharmacology which does not match the properties of either 5HT, or 5-HT2 sites in the brain, These sites are present at a density IO-fold higher than the density of any other serotonergic site in brain membrane homogenates The choroid plexus `251-LSD-binding site appears to represent a new type of mammalian brain serotonergic site. A preliminary report of this work has been published Hartig and Yagaloff, 1985.
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Clin Cancer Res 2002; 8: 54 Lonardo F, Rusch V, Langenfeld J, Dmitrovsky E, Klimstra DS. Overexpression of cyclins D1and E is frequent in bronchial preneoplasia and precedes squamous cell carcinoma development. Cancer Res 1999; 59: 2470 Rusch V, Klimstra D, Linkov I, Dmitrovsky E. Aberrant expression of p53 or the epidermal growth factor receptor is frequent in early bronchial neoplasia and coexpression precedes squamous cell carcinoma development. Cancer Res 1995; 55: 1365 Freemantle SJ, Spinella MJ, Dmitrovsky E. Retinoids in cancer therapy and chemoprevention: promise meets resistance. Oncogene 2003; 22: 7305 Petty WJ, Li N, Biddle A, et al. A novel retinoic acid receptor h isoform and retinoid resistance in lung carcinogenesis. J Natl Cancer Inst 2005; 97: 1645 Dragnev KH, Petty WJ, Ma Y, Rigas JR, Dmitrovsky E. Nonclassical retinoids and lung carcinogenesis. Clin Lung Cancer 2005; 6: 237 Talpur R, Ward S, Apisarnthanarax N, et al. Optimizing bexarotene therapy for cutaneous T-cell lymphoma. J Acad Dermatol 2002; 47: 672 Duvic M, Hymes K, Heald P, et al. Bexarotene is effective and safe for treatment of refractory advancedstage cutaneousT-cell lymphoma: multinational phase II-III trial results. J Clin Oncol 2001 ; 19: 2456 71. Boehm MF, Zhang L, Badea BA, et al. Synthesis and structure-activity relationships of novel retinoid X receptor-selective retinoids. J Med Chem 1994; 37: 2930 Khuri FR, RigasJR, Figlin RA, et al. Multi-institutional phase I II trial of oral bexarotene in combination with cisplatin and vinorelbine in previously untreated patients with advanced non small-cell lung cancer. JClin Oncol 2001 ; 19: 2626 37. Miller VA, Benedetti FM, Rigas JR, et al. Initial clinical trial of a selective retinoid X receptor ligand, LGD1069. J Clin Oncol 1997; 15: 790 Rigas JR, Dragnev KH. Emerging role of rexinoids in non-small cell lung cancer: focus on bexarotene. Oncologist 2005; 10: 22 Yocum RC, Miller VA, Warrell RP, Jr., et al. Longterm follow-up of patients with advanced non small cell lung cancer treated with oral bexarotene. Proc Soc Clin Oncol 2001 ; 20: 267a. 24. Blumenshein G, Khuri FR, Gatzemeier U, et al. A randomized phase III trial comparing bexarotene carboplatin paclitaxel versus carboplatin paclitaxel in chemotherapy-naive patients with advanced or metastatic non small cell lung cancer NSCLC ; . Proc Soc Clin Oncol 2005; 23: 621s LBA7001 ; . 25. Jassem J, Zatloukal P, Ramlou P, et al. A randomized phase III trial comparing bexarotene cisplatin vinorelbine versus cisplatin vinorelbine in chemotherapy-naive patients with advanced or metastatic non small cell lung cancer NSCLC ; . Proc Soc Clin Oncol 2005; 23: 627s LBA7024 ; . 26. Petty WJ, Dragnev KH, Memoli VA, et al. Epidermal growth factor receptor tyrosine kinase inhibition represses cyclin D1in aerodigestive tract cancers. Clin Cancer Res 2004; 10: 7547 Dragnev KH, Petty WJ, Shah S, et al. Bexarotene and erlotinib for aerodigestive tract cancer. J Clin Oncol 2005; 23: 8757 PettyWJ, Dragnev KH, Dmitrovsky E. Cyclin D1as a target for chemoprevention. Lung Cancer 2003; 41 Suppl 1: S155 61. 29. Reddel RR, Ke Y, Gerwin BI, et al. Transformation of human bronchial epithelial cells by infection with SV40 or adenovirus-12 SV40 hybrid virus, or transfection via strontium phosphate coprecipitation with a plasmid containing SV40 early region genes. Cancer Res 1988; 48: 1904 van de Merbel NC, vanVeen JH, Wilkens G, Loewen G. Validated liquid chromatographic method for the determination of bexarotene in human plasma. J Chromatogr B Analyt Technol Biomed Life Sci 2002; 775: 189 Nierenberg DW, Nann SL. A method for determining concentrations of retinol, tocopherol, and five carotenoids in human plasma and tissue samples. J Clin Nutr 1992; 56: 417 Albanell J, Lonardo F, Rusch V, et al. High telomerase activity in primary lung cancers: association with increased cell proliferation rates and advanced pathologic stage. J Natl Cancer Inst 1997; 89: 1609 HermannT, Dragnev KH, Rigas JR, et al. Microarray gene expression analysis reveals molecular activities of bexarotene in in vitro models of non small cell lung cancer NSCLC ; and in tumors of NSCLC patients. Proc Soc Clin Oncol 2005; 23: 679s ; . 34. Govindan R, Crowley J, Schwartzberg L, et al. Phase II trial of bexarotene capsules in patients with advanced non small-cell lung cancer after failure of two or more previous therapies. J Clin Oncol 2006; 24: 4848 Dragnev KH, Petty WJ, Dmitrovsky E. Targeted combination lung carcinogenesis therapy with a nonclassical retinoid and an EGFR inhibitor. Proc AACR 2003; 44: 2701a and bisacodyl.
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Developing in present-day capitalist society "between real human needs and the desires felt by men and systematically stimulated by the agents of monopoly capitalism ; ". `Economic surplus' was Baran's re-elaboration, perhaps for pedagogical reasons, of the classical economists or Marx's `surplus value', and meaning in general terms the portion of output remaining after the consumption necessary for reproduction has been subtracted. However, Baran incorporated important differences and distinguished three kinds: 1 ; the planned surplus was the difference between the optimum output and the optimum consumption of a socialist economy, and was not relevant for a capitalist economy; 2 ; the actual surplus was the difference between actual output and actual consumption, i.e., aggregate savings; finally, and clearly most importantly for Baran, was 3 ; the potential surplus, defined as "the difference between output that could be produced in a given natural and technological environment with the help of employable productive resources, and what might be regarded as essential consumption", whose realisation "presupposes a more or less drastic reorganization of the production and distribution of social output" Baran 1957: 133f. ; . As King 1988: 167 ; points out it is "a hybrid concept involving considerations both of existing capitalist reality and of a more rational socialist future", very clearly revealing Baran's debt to the Frankfurt School: "It is a critical, not solely analytical concept." It includes excess consumption by the upper and sections of the middle classes but not that necessary for government administration ; , output lost due to wasteful organisation and unproductive workers, as well as output that would have been produced but for the effects of deficient aggregate demand. The significance of the latter, Baran 1957: 141-55 ; maintains, was amply demonstrated during the Second World War when large portions were mobilised. In fact, Baran distinguished between `planned potential economic surplus' and merely `potential economic surplus', the difference being between a `rational' and merely `purposeful' employment of currently employed resources. The thought leans on some kind of `underconsumptionism' by which is usually meant the belief that there is a lack of demand because of the restricted purchasing power of workers. A striking thing observable from Howard & King's History of Marxist Economics is how widespread such beliefs were among the early Russian and German Marxists; the non-Marxist Hobson was another influential source of underconsumptionist ideas, not least because it entered into some of Lenin's works, though Baran and Sweezy, according to Brewer 1987 ; , seem unwilling to acknowledge this debt. Sweezy had identified the tendency to underconsumption as the most serious contradiction of the `monopoly stage of capitalism'. Monopolistic prices, as Hilferding had taught, caused monopolies to spread in concentric circles from any point of origin. The equal profit rate of competitive capitalism thereby turned into a hierarchy of profit rates according to the degree of monopolisation of an industry. A generally higher degree of monopolisation raised the rate of accumulation of capital and hence accentuates the falling rate of profit and underconsumptionist tendencies. Under competitive capitalism deficient demand might be counteracted by buoyant investment expenditure but, Sweezy claimed, monopolies rather oligopolies ; lacked the same compulsion to invest in the latest technology. Capital crowded into more competitive areas so as to lower the average rate of profit, which in turn lowered the incentives to invest, and thereby caused depression and strengthened the general tendency toward falling rate of profit and underconsumption. The principal force acting against stagnation was the rising cost of distribution marketing, etc. ; , beyond what was socially necessary, with the consequence that monopoly extra profits were reduced "in many cases to no more than the competitive level", as Sweezy puts it, which must have obstructed any empirical verification or falsification of his general claims on monopolisation , a large number of unproductive consumers were brought into existence, in addition to the indirect boosting of consumption through advertising Sweezy 1942: 270-86 and boniva.
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